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In parallel, SCFAs attenuate neuroinflammation by dampening microglial TLR4/NFB signaling, favoring a shift from proinflammatory M1 toward antiinflammatory M2 phenotypes and lowering the production of ROSpromoting cytokines such as TNF and IL6

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Phase I reactions (primarily oxidation, reduction, and hydrolysis) structurally modify the parent compound but frequently generate reactive intermediates that can be more cytotoxic than the original molecules

J.RonaiZ.1999Regulation of JNK signaling by GSTp.18132134 2

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